Buffalo Bills Fan

They both esp tennis can get tested that day if neg and play.

Both golf and tennis should be open and playing

Don't mind the protesting. Just that people foolish standing too close or near each other. Like President Trump phase's. Phase 1.

I think golf might be one of the safest things to do without a crowd. Shocked they closed golf courses. Def should reopen them.

Post of the day. Agree DR.

Right. Russia daily cases went up third second behind USA, and Germany in testing. Death rate is low for Russia tho. Could be many factors. Agree with some things you said and some others said. Just a pretty complex virus.

Jesus Christ. All people you people never bring any context or anything to post. Besides you're Trump hate. I might agree with you if bring solid info. But not just by pure hate. Hell I like Jrober38 he try to bring info sometimes way too over played at lease respect him. You bring nothing to these boards besides you're hate. I'm a independent. I look at real facts and truth over hateful crap.

Ya they added to there death toll today... +1,290 didn't added from start I think, what they said. CCP blamed USA, Italy so on and so on. They spin everything lol.

lol is this all you guys got? Only thing you can come here with knowledge is sht sandwiches. Jesus you guys eat sht sandwiches? I want no part of you're guys weird food diets. lol

You know i'm a Independent right? Hope they use common sense and not eat crap sandwiches. I think they use common sense over that. Great post facts based rather then opinions based.

https://www.nbcnews.com/news/world/coronavirus-china-announces-jump-death-toll-wuhan-china-n1186006 Nearly 1,300 people who died of the coronavirus in the Chinese city of Wuhan, where the virus was first detected, were not counted in death tolls, state media announced Friday. Officials increased the number of coronavirus deaths by 1,290, bringing the city's total to 3,869. The updated figure follows allegations from the United States that China had failed to alert the world soon enough about the onset of the virus and that their data was skewed. Beijing has dismissed claims that there has been any kind of cover-up. Still low. But there making sure there cover up looks better.

Just wondering if people are looking at the right thing. Coronavirus covid19 can get to cd147, like ACE2. Just wondering why another reason red blood cells. They also help with you're oxygen red blood cells.. Hear ventilators at high doesn't help people. Malaria attacks the blood cells. Same drug hydroxychloroquine helps people fight them both. Just wondering other people thoughts.

This might of what happen 3rdnlng. Just joking around. lol

Ya I mentioned this medication a month ago when was in other Countries. Ya this medication does really help.

Feel for bad President Trump friend died to covid19. People down play or over play it. Just gotta be prepared.

Just happy President Trump prepared for this. If it was a democrat or some not good other person. My biggest worry but President Trump prepared for everything. I'm happy.

Right city's don't have that or much room. Agree with this.

What worries is people get too excited with government opens.. With no social distancing. Acting like a win, should still be a caution with caution with this thing still going on.

Understand all ways. Just looking at all things.

Dr Tedros I wished didn't play favoritism. Problem would be solved way sooner.... Some people I know around here had big problems with. Reminds (people I live) me of dr tedros adhanom ghebreyesus. Favoritism. Rather than fight for real and truth. Would love to have board here replace them like in person knowing would be fun. Have a next door lady kind of like people here on PPP. We get along great.

Agree man with economy.

For me not going to make projections. Everyone seems to get it wrong. Could be or could not. Way too early to talk about July or August for covid19.

Nevermind ..

There is ACE2 receptor along with furin in heart. SARS-CoV-2 and ACE2 Angiotensin-converting enzyme 2 (ACE2) is a membrane-bound aminopeptidase that has a vital role in the cardiovascular and immune systems4. ACE2 is involved in heart function and the development of hypertension and diabetes mellitus. In addition, ACE2 has been identified as a functional receptor for coronaviruses4, including SARS-CoV and SARS-CoV-2. SARS-CoV-2 infection is triggered by binding of the spike protein of the virus to ACE2, which is highly expressed in the heart and lungs4. SARS-CoV-2 mainly invades alveolar epithelial cells, resulting in respiratory symptoms. These symptoms are more severe in patients with CVD, which might be associated with increased secretion of ACE2 in these patients compared with healthy individuals. ACE2 levels can be increased by the use of renin–angiotensin–aldosterone system inhibitors. Given that ACE2 is a functional receptor for SARS-CoV-2, the safety and potential effects of antihypertension therapy with ACE inhibitors or angiotensin-receptor blockers in patients with COVID-19 should be carefully considered. Whether patients with COVID-19 and hypertension who are taking an ACE inhibitor or angiotensin-receptor blocker should switch to another antihypertensive drug remains controversial, and further evidence is required. Acute cardiac injury Reports suggest that the Middle East respiratory syndrome-related coronavirus (MERS-CoV) can cause acute myocarditis and heart failure5. SARS-CoV-2 and MERS-CoV have similar pathogenicity, and the myocardial damage caused by infection with these viruses undoubtedly increases the difficulty and complexity of patient treatment. Myocardial injury associated with the SARS-CoV-2 occurred in 5 of the first 41 patients diagnosed with COVID-19 in Wuhan, which mainly manifested as an increase in high-sensitivity cardiac troponin I (hs-cTnI) levels (>28 pg/ml)3. In this study, four of five patients with myocardial injury were admitted to the intensive-care unit (ICU), which indicates the serious nature of the myocardial injury in patients with COVID-19. Blood-pressure levels were significantly higher in patients treated in the ICU than in those not treated in the ICU (mean systolic blood pressure 145 mmHg versus 122 mmHg; P < 0.001)3. In another report of 138 patients with COVID-19 in Wuhan, 36 patients with severe symptoms were treated in the ICU1. The levels of biomarkers of myocardial injury were significantly higher in patients treated in the ICU than in those not treated in the ICU (median creatine kinase (CK)-MB level 18 U/l versus 14 U/l, P < 0.001; hs-cTnI level 11.0 pg/ml versus 5.1 pg/ml, P = 0.004), suggesting that patients with severe symptoms often have complications involving acute myocardial injury1. In addition, among the confirmed cases of SARS-CoV-2 infection reported by the National Health Commission of China (NHC), some of the patients first went to see a doctor because of cardiovascular symptoms. The patients presented with heart palpitations and chest tightness rather than with respiratory symptoms, such as fever and cough, but were later diagnosed with COVID-19. Among the people who died from COVID-19 reported by the NHC, 11.8% of patients without underlying CVD had substantial heart damage, with elevated levels of cTnI or cardiac arrest during hospitalization. Therefore, in patients with COVID-19, the incidence of cardiovascular symptoms is high, owing to the systemic inflammatory response and immune system disorders during disease progression. The mechanism of acute myocardial injury caused by SARS-CoV-2 infection might be related to ACE2. ACE2 is widely expressed not only in the lungs but also in the cardiovascular system and, therefore, ACE2-related signalling pathways might also have a role in heart injury. Other proposed mechanisms of myocardial injury include a cytokine storm triggered by an imbalanced response by type 1 and type 2 T helper cells3,6, and respiratory dysfunction and hypoxaemia caused by COVID-19, resulting in damage to myocardial cells. Chronic cardiovascular damage A 12-year follow-up survey of 25 patients who recovered from SARS-CoV infection found that 68% had hyperlipidaemia, 44% had cardiovascular system abnormalities and 60% had glucose metabolism disorders7. https://www.nature.com/articles/s41569-020-0360-5?fbclid=IwAR3Ni4Y6EokoleVLOGkmHia8Si03IwBuGV8kRTJjUJEBGS5ULlfAOHb2gHE It's why this is so complex understand both sides. I wonder President Trump when he finds out more about the lab. Hopefully shares with everyone.

Right. But for real anything for her abortion laws in her eyes. Tried to sneak something in stimulus loop hole by supporting that crap. Nancy Pelosi very sad human.